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The following parameters were assessed: age group, sex, mode of trauma, type of injury, place where the trauma occurred, time interval between injury and presentation and the overall morbidity as well as mortality.
Similar differences in age were noted when we assessed age at onset of symptoms (data not shown).
Student's t-tests (p<0.05) or single factor (age) ANOVAs (analysis of variance; p<0.05) combined with post-hoc multiple comparison tests (Tukey-Kramer procedure) were used to compare normalised overlap values across assessed age points.
If maternal dates differed by more than 2 weeks from assessed age, and if early ultrasound data was not available, then assessed age was used.
We also assessed age at diagnosis of diabetes, duration of diabetes, treatment of diabetes, and BMI.
The following individual characteristics were assessed: age, gender, and educational level.
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Children were assessed aged six-seven years via parental-reports of behavioural problems.
Here, we assessed age-related changes in the salivary-specific humoral immunity of DS subjects.
We induced colonic apoptosis using azoxymethane (AOM) in three different aged (6 week, 12 month, and 22 month) F344 rats and assessed age-related differences in induction and potential molecular mechanisms of apoptosis.
Whereas numerous studies assessed histological and physiological changes in the aging olfactory system in mice [e.g. 35] [37], surprisingly few studies so far assessed age-related changes in olfactory performance in mice at the behavioral level.
We assessed age-related differences in the immune response to infection during hospitalization by comparing changes in circulating concentrations of inflammatory (TNF, IL-6, IL-10), coagulation (Factor IX, thrombin-antithrombin complexes [TAT], antithrombin [AT]), and fibrinolysis (plasminogen activator inhibitor [PAI]-1 and D-dimer) systems.
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CEO of Professional Science Editing for Scientists @ prosciediting.com