The phrase "assess binding of" is correct and usable in written English.
It can be used in contexts related to evaluating the attachment or interaction of molecules, such as in scientific or technical discussions.
Example: "In our study, we aim to assess binding of the drug to its target receptor to determine efficacy."
Alternatives: "evaluate the binding of" or "analyze the binding of".
To assess binding of CPS to LL-37 in the presence of human CD14 and LBP, an EMSA assay using native gel conditions was performed [18].
To assess binding of recombinant HcpA for CFH and CFHR-1, ligand affinity blotting techniques and ELISA were employed in combination with intact HcpA and various deletions thereof.
To test this hypothesis, we performed ChIP to assess binding of ESE3 to the promoters in parental and ERG-expressing LNCaP cells.
To test this hypothesis, we performed ChIP to assess binding of ESE3 to the regions of the EZH2 and Nkx3.1 promoters containing the identified EBS in parental and ESE3-kd LNCaP cells.
Finally, to prove that this interaction occurred also in clinical tumor specimens we performed ChIP to assess binding of ERG to the EZH2 promoter in ERGhigh and NoETS samples (Fig. 3G).
Tests to assess binding of H5 and mutants thereof in relation to host range receptor switching should therefore strive to use models that reflect the microenvironment of the luminal surface of the human airway epithelium.
Since TESS predicted that Sp1 binds to MCRE, we assessed binding of cellular Sp1 to MCRE by electrophoretic mobility shift assay (EMSA).
Two mechanisms were assessed: binding of the tracer to melanin or to sigma receptors of melanoma cells.
Next we assessed binding of recombinant SNAP25 and VAMP2 to synatxin1B immunopurified from mouse brains.
To validate the binding of NLX to FLNA, we assessed binding of [3H]NLX to membranes prepared from the human melanoma cell line M2 that lacks filamin and to membranes from its FLNA-transfected subclone A7.
Because AcH4 and H3K4me3 function to enhance gene transcription, in part, by facilitating binding of RNA polymerase II to promoters of regulated genes, we next assessed binding of RNA polymerase II to the TNF-α promoter at both 2 and 4 hrs post LPS treatment in sham and CLP macrophages.
