The phrase "assays we found" is correct and usable in written English.
It can be used when referring to tests or analyses that have been discovered or identified in a particular context, such as scientific research or data analysis.
Example: "In our recent study, we reviewed the various assays we found to determine their effectiveness in detecting the disease."
Alternatives: "tests we discovered" or "analyses we identified".
Based on these GFP reporter assays, we found that NHEJ was clearly impaired when cells lost PARP2 or APLF.
Through both in vivo (Supplementary Fig. 1c) and in vitro (Supplementary Fig. 1d) assays, we found that Int was sufficient to recombine attP and attC sites, as is characteristic of LSRs22,23.
Using in vitro Sm core assembly assays, we found that lowering the intracellular amounts of Gemin3 by either poliovirus infection or small interfering RNA (siRNA -mediated knockdown of Gemin3 resiRNA -mediateded assembly of U snRNPs.
Using nuclease sensitivity assays, we found a progressive decrease in chromatin digestion among pluripotent embryonic stem cells (ESCs), multipotent hematopoietic stem cells (HSCs), and mature hematopoietic cells.
Here, using mammalian two-hybrid assays, we found that transcription repression by ETO was substantially decreased when either zinc finger motif of ETO is deleted or mutated.
Using comparative DNA binding assays, we found that a number of flexible arrangements of Hox, Exd, and Hth binding sites mediate cooperative transcription factor complexes.
Also, using output data of DNA protein interaction assays, we found that 42 of all predicted genes are targets of SOX2, NANOG and POU5F1.
Using quantitative GTPase and fluorescence assays, we found that SulA inhibition resulted in an increase in the apparent critical concentration for FtsZ assembly.
By MTT assays, we found that EB1 siRNAs increased the IC50 values of paclitaxel in all these cell lines (Fig. 2I).
By scratch wound assays, we found that HDAC6 siRNAs remarkably compromised the ability of pancreatic cancer cells to migrate into the wound area (Fig. 3A and 3B).
Using BrdU incorporation assays, we found no evidence for significant alterations in cell proliferation.
