Sentence examples for assays in the form from inspiring English sources
Proliferation assays in the form of CFSE dilution were carried out as described above in the presence or absence of neutralising antibodies to IFN-γ (R&D Systems), TNF-α (eBioscience), isotype control (R&D Systems) or recombinant IL-1 receptor antagonist (IL-1RA) (eBioscience) at concentrations of 0.5, 1.0 and 2.0 ug/ml.
The MDA content, a quantitative measurement of lipid peroxidation, was assayed in the form of thiobarbituric acid reactive substances by the method of Wills [32].
It was assayed in the form of thiobarbituric acid reacting substances (TBARS).
The mean malondialdehyde (MDA) content (μmol/mg protein) was used as an indicator of lipid peroxidation and assayed in the form of thiobarbituric acid-reacting substances (TABRS) [ 28].
The mean thiobarbituric acid reactive substances (TBARS) (μmol/mg protein), a measure of lipid peroxidation, was assayed in the form of malondialdehyde (MDA) using a commercial kit purchased from Cayman Chemical Company (Ann Arbor, MI, USA).
This methodology implies that the quality of a product must be designed instead of assayed in the final dosage form.
Regarding the emulsification assays, in order to form an emulsion, BSs must significantly lower the oil-water interfacial tension.
The dramatic progress made in understanding the haematopoietic system in recent years has come about largely as a consequence of the ability to prospectively identify haematopoietic stem cells on the basis of cell surface antigens [ 17] and as a consequence of the existence of a robust and well-characterised repopulation assay in the form of bone marrow transplantation.
To distinguish between the two possibilities, we assayed in vitro the ability of the fibrillar form of Ure2p 1–79 to seed the assembly of full-length Ure2p.
Therefore, we used medulloblastoma cell lines to assay responses to genotoxic stress in the form of chemotherapeutic agents used to treat medulloblastoma clinically (cisplatin (CDDP), etoposide (VP-16)) and another agent, doxorubicin, previously studied for TP73 induction.
