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Together, both in vivo and in vitro assays argue that instead of causing defects directly on migration, Nf1 inactivation promotes premature neuronal differentiation of a subset of GCPs, leading to abnormal accumulation of differentiated neurons in the ML and EGL.
That 8 of the 16 posterior Hox genes from all posterior paralog groups except Hox12 show an effect in the ingression, elongation, and Wnt signaling assays argue for an extreme redundancy of the system that could further explain the intraspecific robustness of the vertebral formula.
The fact that 8 of the 16 posterior Hox genes from all posterior paralogs groups except Hox12 show an effect in the ingression, elongation and Wnt signaling assays argue for an extreme redundancy of the system that could explain the intraspecific robustness of the vertebral formula.
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They argue.
Go argue.
They even argue about arguing.
And who can argue?
He didn't argue.
We argue "no".
Who can argue?
ECONOMISTS love to argue.
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