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Given that NC2α and SdhB suppressed both tau and polyglutamine toxicity, we assayed the abilities of these genes to suppress other neurodegenerative models: expression of the 42 amino acid isoform of β-amyloid (Aβ42) and an amyotrophic lateral sclerosis-causing mutation in TAR DNA Binding Protein-43, TDP-43Q331K.
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We assayed the ability of co-transformant L 61 to degrade lignin using wood meal derived from Japanese cedar.
We assayed the ability of each defensin to downmodulate CD4, CCR5 and CXCR4.
Subsequently, we assayed the ability of the different SUB gSUBmut:EGFP reporters to rescue the sub-1 phenotype.
Therefore we assayed the ability of Treg or TregKIF to bind to ligands of CD62L, VLA-4, and LFA-1 in an in vitro adherence assay (Fig 4a).
To this end, we assayed the ability of 400 compounds from the Prestwick Chemical Library (PCL), a collection of drugs selected for their biological activity, to increase viability of female flies expressing i CUG 480 RNA in their MBs.
We first assayed the ability of DF-1 fibroblasts to support the growth of MVA in comparison with two other permissive cell types, primary CEFs and BHK-21 cells (Figure 1).
We then assayed the ability of rats with binocular induced glaucoma to entrain their daily locomotor activity to successive 6 hr delays of LD cycles each coupled with 1 log unit decreases in light levels (100, 10 and 1 lux).
We assayed the ability of Tim, Tipin, Plk1, and Aurora A to interact with the chromosome 17 centromere alpha satellite repeat DNA (D17Z1) which can be uniquely amplified by PCR (Fig. 7A).
We assayed the ability of 14-3-3σ 14-3-3σ 14-3-3σtobility of the BCaffectthen.
Initially, we assayed the ability of P. berghei sporozoites to enter liver cells in vitro using an established assay.
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