Sentence examples for assay has indicated from inspiring English sources

The MTT assay has indicated that cell viability has shown an increasing trend on increasing the time of cell seeding from 1 to 3 days.

Bimolecular fluorescence complementation assay has indicated that MLPK interacts with SRK at the plasma membrane and that this interaction is independent of the SCR peptide, suggesting that MLPK might form hetero-oligomers with SRK on the plasma membrane.

Previous studies utilizing the FOX assay have indicated its lack of specificity for hydroperoxides [57].

In these five patients, SnaPshot assay had indicated that a KRAS mutation was present in one case and absent in the remaining five cases.

Our previous studies using GST-GGA1 pull down assay have indicated that CpG ODN, but not GpC ODN, activate TLR9 signaling to induce ARF6 activation.

TEM observation and agarose gel electrophoresis assays have indicated MNPs play the role of driving magnetofectins to the cell surface without entering into the nucleus.

Co-immunoprecipitation assays have indicated that AMSH-2 could associate with SMAD2 and SMAD7 (Ibarrola et al., 2004), but also in this case it is not yet known whether the DUB activity of AMSH-2 is required for the enhancement of TGF-β signaling.

Furthermore, antigenic peptide presentation assays have indicated that ERAP1 can have complex effects on antigenic epitopes depending on the nature of the epitope tested [14], [19].

Northern blot and in situ hybridization assays had indicated that HongrES1 mRNA was specifically expressed in the rat cauda epididymis and restricted to the epithelial cells [15].

The results of water channel activity assays have indicated that both CF expression modes could produce functionally folded mAQP4 M23, while higher Pf values were obtained from D-CF samples.

In fact, immunoprecipitation and immunofluorescence assays have indicated that a complex between RB1CC1, hSNF5 and p53 forms in cell nuclei, and ChIP, quantitative RT-PCR and the luciferase assays for RB1, p16 and p21 have suggested that the coordinated transcriptional enhancements of these molecules are affected by the complex including RB1CC1, hSNF5 and p53.