The phrase "assay exposure" is not commonly used in written English and may not be clear without context.
It could be used in scientific or medical contexts where exposure to a substance is being tested or measured.
Example: "The study aimed to determine the assay exposure of the drug in various concentrations to evaluate its efficacy."
Alternatives: "test exposure" or "exposure assessment".
As shown by MTT assay, exposure to H2O2 (400 µM) for 8 h resulted in significant cell death in all of the cell lines.
In the oscillation activity assay, exposure to 100 μM yakuchinone A for 72 h caused the maximum effect of 27% of H. nana.
Indeed, as showed by TUNEL assay, exposure of TEC for 48 hours to septic plasma induced a significant increase of apoptosis in respect to healthy plasma.
In addition, during the β-hexosaminidase release assay, exposure to the lysate was limited to 30 min, thus reducing any potential toxic effects the M. aeruginosa lysates have on the cells.
In the AB assay, exposure to 6-OH-BDE-47, 6′-OH-BDE-49, or 3-OH-BDE-47 increased the relative fluorescence intensity dose-dependently above the control level (data not shown).
Two exposure scenarios were considered for further modeling: exposure over the course of 24 hr (based on the standard assay exposure duration) and 45 years (a full occupational lifetime).
In our assays, exposure to BAC concentrations of 0.02% for 60 minutes demonstrated no toxicity relative to the saline control.
This raises serious challenges to metrology as nanoscale measurement is required, under experimental conditions, that is, of nanoparticle concentration and dispersion within cell culture medium for in vitro assays (exposure dose) and of nanoparticle concentration within cellular tissue (i.e., dose delivered).
In addition, caspase-3 activity was measured, using a fluorometric assay, upon exposure to bisphenol A (40 nM) alone, leptin (2.5 nM) alone, and the combination of both agents.
Cell viability of primary lymphoma/200 µl/well in 96 well plates was determined by MTT assay after exposure to 25 and 50 µg/ml of C1 for 24 hrs.
Cell proliferation was evaluated by CCK-8 assay after exposure to ROCK inhibitor (Y-27632).
