Sialic acid species were detected separately using a colorimetric assay, accounting for 1% (w/w).
A high level of IL-8 release with IgG1 but not IgG4 control in the solid phase assay accounted for this statistical difference (Table 4).
We found that variation in the four large repeat arrays we assayed account for up to 61percentt of the variance in measured genome size among the accessions.
However, none of these assays accounted for the presence of C-terminal fragments, as these may have been quantified together with full-length (phosphorylated) TDP-43.
Exposure to 17-AG (which has similar potency to retaspimycin HCl in cellular assays) accounted for ~50%% of the total exposure to drug, calculated as the aggregate AUC of parent drug plus the two metabolites.
The DOC released in our assays accounted for only 0.4, 0.8, and 17.1% of the initial carbon content measured by the elemental composition analysis of woodchips, peanut shells, and barley, respectively.
Virus attachment and lectin-binding assay may account for the observed sensitivity of NDV to Lec1 cells.
Upon interaction with airway cells, PM can elicit oxidative stress through alternative pathways; therefore, this assay can account for only a fraction of in vivo PM activity.
Normalization to these internal positive control samples, which are provided with the assay reagents, account for slight differences in assay efficiency (hybridization, purification, binding, etc).. Concentrations of the control transcripts range from 0.125 fM to 128 fM.
It is possible that the use of the standard assay may account for the relatively small impact of CPB on PT in the present study, despite the substantial increase in blood loss observed after CPB and saline infusion.
