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As discussed in the next chapter, TGF-β1 TGF-β1tes bone foregulateshrough enhancement of osteobonet proliformation[ 17], differenthrough[ 18], and chenhancementttractiof[ 19], and alsosteoblastlular matrix and proliferationsynthesis of chondrocyte precursor cells [ 20].
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This distribution changes dramatically after resection of the radial head, as shown in cadaveric studies and as discussed in the following chapter [74].
There exists a continuity between the two, the latter of which is further discussed in the next chapter.
A simplified version of the leukocyte recruitment cascade exemplified for neutrophils is displayed in Fig. 1 (developmental aspects are indicated by footnotes and discussed in the next chapter).
Another low molecular-weight chelator is glutathione (reduced form GSH, oxidized form GSSG) which will be discussed in the next chapter.
This minor difference can be caused by the different morphologies of Au nanostructures influencing the transport of free carriers in Au nanolayers after annealing, which will be discussed in the next chapter.
The clinical implications of this model are discussed in the next chapter.
However, the antipsychotic action of this antagonist effect remains questionable and will be discussed in the next chapter.
This suggests that the DNA methylation pattern of MRL/MpJ mouse might have retained some embryonic features and the possibility will be discussed in the next chapter.
While HD6 so far only exhibited little bactericidal potential in vitro but employs additional defense strategies, which will be discussed in the next chapter, investigations on HD5 exposed an effective killing capacity against various bacteria (Ericksen et al, 2005).
We modified this hypothesis as discussed in the next section.
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