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Studies in Xenopus oocytes and mammalian cells provide evidence that Casp2 is metabolically regulated, acting as a sensor to changes in NADPH levels resulting from altered flux through the pentose-phosphate pathway (PPP).
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Our data support a model in which Ng targets CaM within the dendritic spine, and acts as a sensor to local Ca2+ changes.
Such a polybasic region may be able to specifically recognize negatively charged phospholipids, but might also act as a sensor for changes in transmembrane voltage.
Due to the sensor sensitivity to changes in pH at 480 nm excitation data presented are normalized to 1 − (F430/ F0) as described previously [24].
The developed capacitive sensors respond to changes in solute concentration as a function of distance from the membrane surface.
The reversible reactions from F6P to Mannitol6P and FBP to G3P can be used as a sensor reaction to determine the change in redox status in the cytosol.
Sensors that respond to changes in light and the motions of passers-by will trigger electronic devices that cause giant snail shells, metallic bowls and other containers to produce sound as if they were loudspeakers.
Dependence of Sirt 1 on NAD+ levels infers that it acts as a key sensor for changes in metabolism.
As such, HIF-1 is considered as the master sensor that orchestrates cellular responses to changes in oxygen homeostasis.
Recent findings have suggested that stromal interaction molecules (STIMs) function as the Ca2+ sensor to detect changes of Ca2+ content in the intracellular Ca2+ stores.
The GK protein acts in pancreatic islets as a "glucose sensor" that couples fluctuations in the blood glucose concentration to changes in cellular function and insulin secretion.
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