Specifically, we expected that the making of a commitment to save energy (Hypothesis 1) as well as receiving feedback on energy use (Hypothesis 2) would lead to changes in behavior as well as environmental concern compared to a control group.
One possible explanation for this late-life decline in giving relates to changes in physical health, as we argued in our physical health hypothesis.
As for hypothesis 1, baseline variables that are predictive of outcome and/or differ between groups will be included as covariates in the analyses for hypothesis 2. Hypothesis 3 involves the correlation of changes in beliefs and coping to changes in the outcome variables.
As Farrell (2013) observes, most of the crime drop hypotheses are crime generic, attributing changes in crime to changes in the number or motivation of offenders.
The hypothesis further predicts that plants can change their defenses in response to changes in nutrients.
Our results negate this hypothesis as the expression of CCM-genes under long term acclimation (steady state conditions) was insensitive to changes in experimental conditions.
To examine the hypothesis, we did DNA methylation analysis by pyrosequencing, which was recently developed as a quantitative technique to detect changes in methylation patterns [29], [30].
Our various 2020 model predictions can serve as testable alternative hypotheses concerning changes in species distribution and richness that will play out in the next 10 years.
As noted, ACA's Medicaid expansion and premium subsidies have boosted insurance coverage, so one way to test the job-killer hypothesis is to plot changes in employment growth by state against changes in insurance coverage (this figure shows 2013-2015 changes, but the results are the same for one-year changes).
Consistent with our hypothesis, pathological changes in the basal ganglia, as well as extra-nigral structural alterations, particularly in the cortex, predict motor deficits in this model.
This further supports the hypothesis that changes in these markers may act as pharmacodynamic markers for VEGFR inhibition.
