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Using pore size distributions determined from LLP, a mechanistic mathematical model grounded on the principle of particle retention by size exclusion closely predicted measured virus retention performance, both as a function of virus size and of membrane pore size.
We used Poisson regression with offsets to model the natural logarithm of AGI episodes per person-day of at-risk follow-up as a function of virus exposure.
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Experimental infections of passerine species, e.g. house sparrows, European starling [18], [19], zebra and house finches [19] also characterized these birds as vulnerable, but their susceptibility differed as a function of the virus isolate used.
The currently recommended regimen is based on the use of pegylated interferon (PEG-IFN) combined with ribavirin (RBV) for 24 to 72 weeks as a function of the virus genotype, the virologic response in the course of treatment, and the patient's tolerance [ 8].
A feature of adenovirus infection is that functional expression level of the recombinant enzyme is modulated as a function of the amount of virus used.
We calculated the probability of an MDV outbreak as a function of the initial amount of virus in the barn.
Gene delivery via the NH2-SAM was further characterized as a function of retrovirus coating time, virus concentration, and cell seeding density.
Results from analysis of variance of (a) viral RNA concentration in the bodies (thorax+abdomen) of infected mosquitoes and (b) mean virus titer (determined by FFA in Vero cells) in the head/legs of mosquitoes with a disseminated infection as a function of mosquito isofemale families, virus isolates, experimental blocks, and their interactions.
The table shows the nominal logistic regression for the proportion of (a) mosquitoes with detectable viral RNA in their bodies (thorax+abdomen) and (b) infected mosquitoes (excluding uninfected) with a disseminated infection in their head/legs (determined by FFA in Vero cells) as a function of mosquito isofemale families, virus isolates, experimental blocks, and their interactions.
A major characteristic of this approach is that functional levels of CYP are easily modulated as a function of the number of infecting virus particles (Fig. 31).
In these simulations we also investigated three hypothetical contamination scenarios at stopover sites on migrations, as a function of the number of infectious virus units present in halt meshes.
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