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Patients requiring artificial ventilation due to primary failure of respiratory muscles and those who required artificial ventilation due to brain injury were also excluded.
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To prevent this latter effect, the increase in dead space ventilation due to the artificial nose must be compensated for by an increase in minute ventilation.
One patient died 1 month after study inclusion due to cardiac failure; the second death occurred after a 5-month stay at the intensive care unit with prolonged artificial ventilation, hemofiltration, recurrent infections, and finally death due to multi-organ failure after drainage of a retroperitoneal hematoma.
The study encompassed 29 patients of both sexes who were admitted to the intensive care unit due to sepsis and required artificial ventilation as the result of acute respiratory failure.
Our results support previous suggestions that artificial ventilation can exert a direct influence on the IAP due to increased intrathoracic pressures that are then transmitted to the abdomen [1, 2, 7].
Our results support previous suggestions that artificial ventilation can exert a direct influence on the IAP due to increased intrathoracic pressures that are then transmitted to the abdomen [ 1, 2, 7].
In particular, the fact that similar results were obtained using different anaesthetics (urethane vs. halothane) and methods of ventilation (spontaneous breathing vs. artificial ventilation with O2/N2O) argues that these findings are not due to elevated or depressed levels of systemic oxygenation.
The postoperative course was delayed due to difficulties in weaning the patient off artificial ventilation given the existing pulmonary weakness.
A ventilator has the function of providing the patient with artificial ventilation.
Due to dry medical gases, humidification of the inspired gases is essential during prolonged artificial ventilation in endotracheally intubated patients.
Positive pressure ventilation is a method of artificial ventilation.
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