The phrase "arterial preparations" is correct and usable in written English.
It can be used in medical or pharmaceutical contexts to refer to products or formulations related to arteries or arterial health.
Example: "The study focused on the effectiveness of various arterial preparations in improving blood flow."
Alternatives: "vascular formulations" or "arterial products".
Here, we addressed this concern by alternative, more quantitative approach, i.e. by comparing Panx1 gene expression levels in endothelium-intact and endothelium-denuded arterial preparations.
When normalized to vessel tension, contractions induced by K+ (60 mM) were similar in WKY to our previous findings in arterial preparations of adult male Wistar rats (21).
All arterial preparations were kept in physiological salt solution (PSS, pH 7.4) containing (mM): NaCl 119.0, NaHCO3 25.0, glucose 11.1, CaCl2 1.6, KCl 4.7, KH2PO4 1.2, MgSO4 1.2, and aerated with 95% O2 and 5% CO2 at 37°C.
The impact of oxygenation on H2O2 accumulation was examined in experiments conducted with buffer exposed to air, because isolated arterial preparations are conventionally maintained in buffer gassed with O2 at levels well above the physiological range.
In large arterial preparations e.g. rabbit and porcine aorta exposed to 21% O2, O2 tension was found to fall and reach very low levels in media of the vascular wall [ 17].
The large arterial rings were equilibrated in a resting preload of 4.905 mN/mm [ 16], while the small arterial preparations were normalized so that the internal diameter of the vessel was set at 90% of that obtained when exposed to intraluminal pressure of 100 mmHg in the relaxed state [ 15, 17].
In vitro arterial preparation was used to study the reactivity of mouse aorta and first-order branches of the mesenteric resistance artery as described previously [ 21].
One basilar arterial preparation obtained from one animal was used for each experiment, and therefore the number of experiments (n value) equals the number of rabbits.
Most evidence for the repolarization hypothesis is, however, derived from studies in the canine arterial wedge preparation, in which BrS is modeled through pharmacological maneuver with potentially nonspecific effects (1, 12).
During graft preparation, arterial blood pressure was increased stepwise in intervals of 10 to 15 minutes by infusion of noradrenaline and lowered thereafter to baseline levels.
Only ET-1[1-32 ET-1[1-32 ET-1[1-32al activities, which were first discovered using an in vitro arterial system upon the preparation of the peptide in vitro (7).
