Sentence examples for arrest level from inspiring English sources

Inversely and importantly, for any given cell arrest level, prevention is, on average, superior to comparable post-diagnostic interventions, even when including cases where prevention initially fails, and resection and additional therapy are needed.

The Resuscitation Outcomes Consortium (ROC) Epistry-Cardiac Arrest database was queried for all patients <19 years old from December 2005 to November 2011 in the Toronto region for age, sex, event characteristics, underlying conditions, cause of the cardiac arrest, level of EMS care, time to EMS arrival, scene time, return of spontaneous circulation (ROSC) and survival to hospital discharge.

Hereafter, we refer to σ as the treatment intensity (applied once every cell cycle of 4 days), while the corresponding daily arrest level to non-resistant cells is approximated by σ/4.> -wrap-foot> We first study preventive interventions where a patient has a high risk of developing a cancer and/or a biomarker that indicates the probable presence of a cancer.

The data also showed a drastic decrease in arrest levels since the mid-1990s: there were an estimated 2.9 million such arrests in 1996, when the population of those under 18 was smaller than it is today.

In contrast, post-diagnostic intervention improves only marginally beyond daily arrest levels of about 0.3%.

Importantly, for both thresholds of tumour excision, subsequent cancer cell arrest levels beyond approximately σ = 1.5% make little difference in terms of tumour growth, since virtually all of the sensitive cells post-excision will be arrested or killed by the measure beyond this level, leaving uncontrollable resistant cells to grow and repopulate the primary tumour site and/or metastases.

Daily arresting level assumed to be 0.25%.

This effect consists of a transient reduction in myocardial performance from pre-arrest levels, which is commonly observed after ROSC [ 14].

In engineered H1299 cell systems with ectopic wtp53, it was previously shown that p53 levels can be a determinant of arrest (lower levels) versus apoptosis (higher levels).

Deficiency in RPA1 led to S phase arrest, increased level of DNA damage in S phase and increased chromosomal instability [35], [36].

Endotracheal intubation was done for usual indications such as respiratory arrest, deteriorating level of consciousness, rising PaCO2 despite maximal pharmacological treatment and deteriorating acidemia.