Sentence examples for arrest duration from inspiring English sources

It can also be seen that the arrest duration distribution was approximately exponential in shape.

The mean arrest duration across mice was 42.2±6 s (n = 16) which is comparable to the mean arrest duration in ataxin-3 mice and ligand knockout mice which ranged from 41 s to 66 s [9], [11], [26], [27].

This is consistent with cholinergic systems influencing arrest initiation circuitry without being part of the circuits determining arrest duration.

Doses of physostigmine that altered arrest frequency should also have altered arrest duration if this circuit controls behavioral arrests.

Since perturbations of muscarinic transmission altered arrest frequency but not lifetime, our findings suggest cholinergic systems influence arrest initiation without influencing circuits that determine arrest duration.

This is indicated in Fig. 2A by the range of arrest durations which is plotted along with the mean arrest duration for each mouse.

To gain insight into the processes governing expression of behavioral arrests, we examined the distribution of arrest durations.

Although altered cholinergic transmission changed the number of arrests, it did not change the distribution of arrest durations.

Two other genes scored with this phenotype, PMAIP1 and NPC2, and these were also notable for very short mitotic arrest durations (Figure 7A).

Despite this, the distribution of arrest durations was not significantly different from that following ACSF or Neo alone (Fig. 7C; K-S test p>0.05).

However, neither the mean bout duration was altered (p>0.05 2-way ANOVA), nor was the distribution of arrest durations (Fig. 7C) altered, even though the number of arrests was increased (K-S test P>0.05).