The recruitment of leukocytes to areas of inflammation is a crucial process for the pathogenesis of IBD1,2.
Many cytokine genes were more highly expressed in the focal areas of inflammation.
Pathologically, multiple scattered areas of inflammation, called plaques, are found within the white matter of the central nervous system.
In other cases, however, the granulomas produce large areas of inflammation and scarring that can interfere with an organ's normal functions.
When infected stem cells were injected into mice, they recruited macrophages to the infection site, which then take up the bacteria and accumulate to form areas of inflammation called granulomas.
The activation and recruitment of microglia and monocytes into areas of inflammation may play a critical role in the pathogenesis of acute brain injury.
Cell adhesion molecules govern leukocyte-endothelial cell (EC) interactions that are essential in regulating leukocyte recruitment, adhesion, and transmigration in areas of inflammation.
The white blood cells are able to migrate through blood-vessel walls in areas of inflammation or infection, where they may phagocytize foreign material such as bacteria.
Aortic valve stenosis and atherosclerotic disease have several risk factors in common, in particular, hypercholesterolemia. Histologically, the diseased valves appear to have areas of inflammation much like atherosclerotic plaques.
Multiple Sclerosis (MS) is a disease of the central nervous system characterized by multiple areas of inflammation and demyelination in the white matter of the brain and spinal cord.
Although local anesthesia is commonly applied for pain relief, there are several issues such as its short duration of action and low effectiveness at the areas of inflammation due to the acidic pH.
