Sentence examples for area of nox from inspiring English sources

The area of NOX enzymes activation in CX3CR1+/−EGFP cells was 11 and 14%%, respectively.

In CD4+YFP cells, the area of NOX enzymes activation was low at peak of disease, amounting to only 3%%.

We quantified the contribution of specific cell types to the total area of NOX enzymes activity in the CNS using the cellular markers described above.

In untreated RRMS patients, the area of NOX activation was 18.3 ± 2.5 %, in mice at peak of EAE, the NOX activation area was 18.5 ± 3.5%%.

In healthy humans and healthy mice, CD11b+ monocytes were not activated after isolation, i.e., the area of NOX activation amounted to 3.4 ± 0.6 and 3.4 ± 1.9 %, respectively.

Surprisingly, we found that astrocytes (sulforhodamine 101-labeled cells [ 3]) accounted for 26.4 ± 4.4 and 34.2 ± 5.6%% of total area of NOX enzymes overactivation at onset and peak of EAE, respectively (Fig.  4d).

The area of NOX-specific activation in acute lesions in the brainstem was significantly enlarged, both at the onset of clinical signs, with a mean of 5 ± 1.5 % in the field of view (300 × 300 µm), and at the peak of disease, with a mean area of 14.9 ± 5.8 %, as compared to healthy animals, with a mean area of 2.2 ± 0.9 % (Fig.  1e).

The result in this study weakly suggests that people living in areas with high levels of NOX may even develop allergy to a lesser degree then people living in unpolluted areas.

All values of NOX activation area represent mean values acquired over 30 120 cells per individual.

In the last decade, considerable expansion of the traditional area of environmental catalysis (mainly NOx removal from stationary and mobile sources, and VOC conversion) has also occurred.

The remaining percentage of NOX enzyme activation area that was not associated with labeled cells in this model (12.85 % at peak, 35.46 % in onset of EAE) is probably due to unlabeled macrophages, microglia and/or astrocytes.