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Consequently, there are variable levels of parasite/cell interaction during in vivo T. cruzi infection, which is also dependent of the infecting strain.
There are variable levels of openness in data sharing.
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Due to constraints on primer design resulting from the high level of multiplexing, it is unavoidable that there will be variable levels of amplification for different targets.
In contrast, samples refractory to staurosporine displayed elevated Jak/Stat pathway activity whereas there were variable levels of Jak/Stat pathway activity across a range of etoposide induced responses (Figure 5A –5A).
There was variable level of agreement between methods.
Levels of pS6 were variable, whereas levels of p-AKT were less variable between individual mice (individual mice were identified by numbers shown above each blot).
Of particular interest were variable expression levels of NANOG among blastocysts, although the implication of it is unknown at present.
For immigrants, the relationship between deprivation and psychiatric care use was variable across levels of MD.
Cytotoxic T-lymphocyte (CTL) responses are associated with variable levels of HIV-1 infection control [1] [3], yet these responses are unable to clear the virus.
In plasma and BALF TNF-α could not be detected, whereas variable levels of IFN-γ were found at pre- and post-infection times.
Such mutations can confer resistance to macrolide-lincosamide-streptogramin B antimicrobial drugs and are associated with variable levels of in vitro resistance.
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