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Indeed, several V1Rs and V2Rs are shown to respond to pheromonal substances [6], [22].
Cells with constrained cell shape are shown to respond more slowly to reorienting stimuli.
In transfected cell systems, these receptors are shown to respond to relaxin by causing an elevation of intracellular cAMP, presumably through a G-protein-dependent activation of adenylate cyclase [ 32].
Although endothelial cells of different sources are heterogeneous, human umbilical vein endothelial cells (HUVECs) are shown to respond to LPS and are extensively used for exploration of the mechanism implicated in endothelial cell injury [ 4].
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Endothelial cells also actively express TLR4 and have been shown to respond to inflammatory stimuli like LPS in vitro44,45,46.
Thus, neurons in monkey caudomedial auditory cortex and in human STG have been shown to respond to tactile stimuli47,48,49,50.
Moreover, different genotypes have been shown to respond differently to placebos eg, in studies of social anxiety, depression, and pain.
Recently, Piezo1 channels were shown to respond to fluid shear stress and are crucial for vascular development.
Additionally, many genes encoding transcription factors, cytochrome P450s, glycosyltransferases, methyltransferases and transporters were shown to respond to MeJA elicitation.
Distinct cell types have been shown to respond to activated Ras signaling in a cell-specific manner.
PTFE is shown to respond to uniaxial deformation by undergoing a crystalline phase transition that is previously believed to occur only at very high hydrostatic pressure.
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