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The similarities of the clinical phenotypes of BP-I and BP-II, suggest that BP-I and BP-II are likely to exist in a disease spectrum.
Furthermore, such behaviours are likely to exist in a continuum, with patterns being unique between individuals [ 32].
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However older topics, like Shakespeare, are likely to exist in both places.
Other mechanisms are likely to exist in animal cancers in which BPV-2 infection may play a central role.
In the near future, patent-mapping opportunities are likely to exist in three places.
These mutations are likely to exist in the genome of some cancer types.
Further mechanisms for nuclear and cytoplasmic β-catenin accumulation are likely to exist in esophageal carcinogenesis.
Similar scenarios are likely to exist in other species.
Similar misassumptions and misallocations are likely to exist in other nations with large urban slum populations.
Other isoform-specific mediators of ethylene responses, besides RTE1, are likely to exist in plants.
Similar situations are likely to exist in other systems as well.
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