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Previous studies indicated that CNVs are in variable linkage disequilibrium (LD) with flanking SNPs [ 40- 42].
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Both SNPs are in strong linkage disequilibrium (D′=0.926, r2=0.798).
The variable parameter for this program is the total number of loci that are in linkage equilibrium with each other.
A variable tandem repeat genetic variant in intron 2 of IL-1RN which is in strong linkage disequilibrium with the C+2018T has also been studied.
One of the difficulties with finding an association with any SNP in the MHC region is the strong, but variable, linkage disequilibrium.
These pairs involved all the loci studied except the two less variable ones (MTIC126 and MTIC143) which were in linkage equilibrium with all the other loci.
The BsmI polymorphism was in linkage disequilibrium with a candidate translational control site, the variable length poly (A) sequence in the 3′ untranslated region.
We did not include SNPs associated with variable fluorouracil metabolism, as these do not appear to be in linkage disequilibrium with the variants we examined, minimizing unmeasured confounding.
They are said to be in linkage disequilibrium [85].
Two variables have been identified as linkage variables and no variable as autonomous variable.
The variants are not in linkage disequilibrium.
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