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Increased levels of peroxynitrite are found in active multiple sclerosis lesions [64].
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Increased glutaminase (glutamate-producing enzyme) expression in activated immune cells was found in active MS lesions [3].
Furthermore, in these patients, the levels of MPO-ANCA were dependent on disease activity, with high levels being found in active phases of the vasculitic disease [ 15].
Intense angiogenesis has been found in active pulmonary tuberculosis lesions.
The presence of active TrPs indicates sensitization of muscle nociceptors in both TMD and FMS since high levels of neuroactive mediators [12, 13] and lower pressure pain thresholds [34 37] have been found in active TrPs.
The presence of active TrPs indicate sensitization of peripheral muscular nociceptors since high levels of chemical mediators [33, 34] and greater mechanical hypersensitivity [35, 36] have been found in active TrPs.
Although, both residues, T and S, have been found in active ATPases, the detailed catalytic consequences of their preference in most NLR proteins remains undefined.
The silencing histone mark H3K9-me3 also bound to methylated and unmethylated rRNA promoters to an equal degree, indicating that H3K9-me is found in active chromatin [24].
With the exception of I571C only hydrophobic residues were found in active variants.
Significantly higher (P < 0.05) NFATc1-positive inflammatory cell aggregates were found in active RA tissues than in healthy synovial tissue.
In the OMDM, a significant increase was found in active caspase 8 while both active caspase 3 and 8 were significantly increased in OSDM in NB21 hearts.
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