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SAMD9 and SAMD9L are expressed ubiquitously in human tissues, but they are expression at lower levels in neoplasia.
The two factors with the strongest associations with other data types in humans are expression breadth (the number of tissues one protein is expressed in) and phyletic age (defined by the evolutionarily most distant species where homologs can be found) [ 1, 2].
With regard to inflammation, it is suggested that the initial triggers for activation of vasculature are expression of histamine receptors.
The basic intuition is that more sophisticated products are by definition less needed, as they are expression of a more complex need.
A finer division usually makes the descriptor more discriminative but sometimes, for example when there are expression variations, will bring some problems.
The first steps towards understanding how these biosynthetic enzymes cooperatively interact to produce the depsipeptide product are expression and isolation of each enzyme to probe its specificity and function.
Key ingredients of the model are: expression of the residual in terms of mass contents, consistent lumping of the storage terms in the residual and algorithmic (tangent) matrix, consistent integration rules, the use of a minimum relative permeability and a time marching scheme based on trapezoidal integration.
In this letter, we have extended to Equation 1 an EvcHB method presented in[21, 22, 23, 24, 25, 26, 27, 28], performed symbolic computation, and obtained a new auto-Bäcklund transformation and a families of the exact, analytic soliton-type solutions, two-solitary wave solution for Equation 1, which are Expression (24) and (38).
Such still-unknown functions thus would not be dependent on lamin gene expression pattern, suggesting the presence of other lamin functions that are expression dependent.
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These are expression-based, illumination-based, gender-based, and age-based experiments.
Yet not necessarily all lamin gene functions are expression-dependent.
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