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These proteins are believed to trigger the death of brain cells which make molecules like dopamine needed for movement and other functions.
Heavily hydroxylated chains of these polymers are believed to trigger the alternative pathway.
The uptake of oxLDL and concomitant activation of the O2-producing NAD(P H oxidase and/or oxLDL as a self-contained emitter of O2 are believed to trigger these malfunctions.
Other toxic pollutants are believed to trigger asthma attacks.
This results in the formation of new epitopes which are believed to trigger the primary immune response by which the autoantibodies against tTg develop.
Increased levels of intracellular Ca2+ are believed to trigger Cn activation and to be a key regulator of a variety of physiological and pathological processes [10], [14], [15].
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Wind is believed to trigger alveolar weathering in the heterogeneous sparite.
In addition, they also display the potential to carry therapeutic agents to reduce cerebrovascular inflammation associated with CAA, which is believed to trigger hemorrhage in CAA patients.
Hypobaric pressure is believed to trigger vasodilation due to a decrease in the atmospheric oxygen concentration that can cause a headache.
In this respect, the hyperpolarizing effect of K+ channels on the plasma membrane was thought to provide a driving force for the influx of Ca2+, which was believed to trigger cell proliferation [68], [69].
Chronic estrogen exposure is believed to trigger the transformation to hyperplasia followed by EC [ 16].
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com