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As a result, the inbred mouse panel approach successfully identified an unsuspected genetic factor underlying susceptibility to acetaminophen (paracetamol) hepatotoxicity in humans.
The statistical approach successfully identified significant effects and their interactions, and provided insights into the role of codon-usage effects in expression of viral structural protein.
This approach successfully identified synapse defective (syd) genes that function in active zone formation and synapse morphology [21] [22], neuronal polarity [20], [23] and vesicle transport [24].
This approach successfully identified 62,042 putative SNPs.
This approach successfully identified the deletion in Df 3L BSC27.
These findings established that our C. elegans approach successfully identified genes involved in KS/nIHH.
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The approach successfully identifies all four known polymorphs within this class, as well as a large number of other low-energy structures.
The NMF approach successfully identifies the positioning for all motifs in the sets with T ≥ 300 (r ≥ 0.80), with nearly perfect reproduction for T ≥ 3000 (r ≥ 0.88).
This indicates that the regional approach successfully identifies autosomes with genetic variation attributable to the trait and that genetic variance is not correlated to the length of autosomes as seen by Yang et al. [ 24].
Experimental results suggested that those approaches successfully identified all primary collusive clique scenarios in all selected datasets and thus showed the effectiveness and stableness of the novel application.
These approaches successfully identified genes required for saxitoxin-synthesis in the two transcriptomes.
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Justyna Jupowicz-Kozak
CEO of Professional Science Editing for Scientists @ prosciediting.com