Sentence examples for any virulence from inspiring English sources

The microorganisms that may cause disease in humans and animals but are unlikely to be a serious hazard to laboratory personnel, the community, animals or the environment are called risk group 2. However, the strain E. coli BL21 DE3 pLysS has been known to be non-pathogenic and does not carry any virulence factor or pathogenic mechanism causing infections (Chart et al. 2000).

However, even with this under-prediction, we notably never observe a statistically significant association of virulence factors with regions outside of GIs for any virulence factor class.

Nevertheless, it was interesting that 8% of the isolates (12/150) from infection cases did not reveal any virulence markers under investigation.

It appears that these isolates, though devoid of any virulence factors as probed by us, were able to localize in the deeper tissues of a compromised urinary tract where scarcely any drug reaches in therapeutic quantum.

However, none were unique to EAEC 042, and all had homologues within the non-pathogenic laboratory strain E. coli K-12, suggesting EAEC 042 does not possess any virulence factors which require the Tat system for secretion.

While such an analysis is still limited by the scope of bacterial genome sequences and virulence factors currently available, any virulence factors observed to be present in pathogenic strains from diverse bacterial genera, with no detectable homologs in non-pathogenic strains of the same genera, are considered good candidates for being classified as pathogen-associated.

Furthermore, our approach is hypothesis-free and allows us, in contrast to the above methods, to identify new proteins associated with pathogenicity even if they do not have any similarity to any known virulence factor.

We did not identify any relationship between the prevalence of any studied virulence factors and the difference of GC incidences in Bhutan, Myanmar, and Vietnam.

Also unlike O157, the dominant emergence of ST131 does not seem to be linked to any increased virulence phenotype (Johnson et al. 2012) or any set of specific or unique genetic loci (Clark et al. 2012) other than dissemination of CTX-M 15.

However, the V. parahaemolyticus strains isolated during this outbreak were pathogenic to fish, but they did not possess any detectable virulence gene.

The phage does not encode any known virulence factors and deletion of the entire phage was without effect in several laboratory models of meningococcal pathogenesis [20].