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In conclusion, in this increasingly complex environment, health sciences have to continuously and progressively develop methods and tools to decrease the uncertainty.
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Haematomas are recognised by the characteristic 30 45-HU 30 45-HUion of fresh blood, which becomes even more hyperdense (HU > 60) attenuation hofrs freshclotting, and progressively develops inhomogeneity and fluid-fluid levels from mixing of clotted regions and serum from haemoglobloodysis.
However, when the same train protocol was performed in the presence of 100 µM Cu-Phen, the current markedly increased (by 3.45-fold; n = 6, p<0.01) and progressively developed an instantaneous component (Figure 6C).
The 3xTg-AD mouse line harbors a knock-in mutation for presenilin 1 (PS1M146V) and transgenes for the amyloid precursor protein (APPswe) and for tau (tauP301L) and progressively develops both an Aβ and tau pathology in the cortex and hippocampus [49].
The triple-transgenic Alzheimer (3 × Tg-AD) mouse expresses mutant PS1M146V, APPswe, and tauP301L transgenes and progressively develops plaques and neurofibrillary tangles with a temporal- and region-specific profile that resembles the neuropathological progression of Alzheimer's disease (AD).
Triple transgenic (3xTgAD) mice have mutations in APP, PS1 and tau and, as a consequence, progressively develop both Aβ plaques and neurofibrillary tangles with a temporal and regional-specific profile characteristic of human AD (Oddo et al., 2003).
Students progressively develop and receive weekly feedback on a concept note to support a funding proposal addressing a research question of their choosing.
In the early phases of DR, alterations of retinal capillaries and focal aneurismal deformations progressively develop, very similar to what was observed in retina exposed chronically to PGF or in retina from GK rats.
Patients progressively develop both physical and functional deficits including multiple seizure types, cognitive and physical decline, malnutrition and psychiatric features.
Triple-transgenic AD (3xTg-AD) mice, which progressively develop both amyloid and neurofibrillary tangle pathology, were vaccinated thrice with HSVIEAβCMVIL-4, or a set of control amplicon vectors.
These mice progressively develop both amyloid and tau pathology, which are both associated with synaptic dysfunction.
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