Exact(21)
To illustrate the observed characteristics of tumours diagnosed in different breast density groups, we plotted histograms of the relative frequency of quintiles of tumour size according to breast density quintiles (using quintile categories due to inadequate power to assess by decile) and mode of detection (screen-detected or interval cancers).
Therefore, in addition to including the variables in the first model, the second model included the following variables: tumor size (continuous), lymph node metastasis (continuous), ER status (categorical), PR status (categorical), grade (categorical), and mode of detection (binary: screening-detected versus non-screening-detected cancer) [ 27, 28].
Breast cancer mortality models for ER positive and ER negative tumours were constructed using Cox proportional hazards, adjusted for known prognostic factors and mode of detection (symptomatic versus screen-detected) [ 9].
Histograms of tumour characteristics (size and mode of detection) according to breast density highlighted the shift from predominantly small screen-detected tumours in women with low breast density to larger screen-detected and interval cancers in women with high breast density, for both Cumulus and AutoDensity classifications of breast density.
The rate and site of recurrence and mode of detection were analysed.
Table 4 displays the relation among pathological characteristics, biological marker expression, and mode of detection in the study group.
Similar(39)
Two detection schemes can be used: a "dip-and-dry" mode of detection and an in situ sensing mode are demonstrated for several compounds, including cresyl fast violet (CFV), brilliant cresyl blue (BCB), p-aminobenzoic acid (PABA) and 3-aminopyrene (3AP).
Two detection schemes can be used: a 'dip-and-dry' mode of detection and an in situ sensing mode are demonstrated for several compounds, including cresyl fast violet (CFV), brilliant cresyl blue (BCB), p-aminobenzoic acid (PABA) and 3-aminopyrene (3AP).
Breast cancer mortality models for oestrogen receptor (ER) positive and ER negative tumours were derived from these data using Cox proportional hazards, adjusting for prognostic factors and mode of cancer detection (symptomatic versus screen-detected).
The aims of this study were to examine prognosis after CBC in relation to time interval between BC1 and BC2, mode of detection of BC2, and treatment for BC1.
Previous studies indicate that prognosis after CBC could be associated with age, time interval between BC1 and BC2, mode of detection of BC2, and adjuvant treatment for BC1 [ 4, 15- 17, 19].
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