A maternal history of BEN was associated with a significant increase in systolic blood pressure of 6.7 mm Hg, paternal BEN with a non-significant increase of 3.2 mm Hg, and both parents affected, a significant increase of 9.9 mm Hg.
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There was a similar relation of kidney cortex width and parental history of BEN with pulse pressure; however, no association with diastolic blood pressure was found.
In order to estimate the association of minimal kidney width and parental history of BEN with blood pressure based on repeated measurements, we applied linear mixed models.
My father objected that in forty years of medical practice he had never seen an illness with quite the same profile as BEN, with kidneys so shrunken, fibrosis so severe, and such an advanced state of disease with no hypertension.
The lack of association of either kidney cortex width or a parental history of BEN with diastolic blood pressure may either stem from the larger individual variance of this parameter or may indicate that there is no such link.
