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Two recent studies have analyzed this subject.
In the present study we analyzed this subject.
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Even though it would have been perfectly possible to analyze this subject with the present approach, we decided, for reasons of homogeneity in the inter-subject comparison, to focus on subjects with identical stimulus sequence.
We have analyzed this RNA from selected subjects using a comprehensive and standardized human array, the U133A/B Affymetrix GeneChip set (Iacobuzio-Donahue et al. 2003).
Why? Legions of pundits, writers and insomniac callers to late-night radio have analyzed the subject.
Moreover, we observed that the nature of the dystrophin gene mutation did not affect the levels of both CD133+CXCR4+CD34- and CD133+CXCR4+CD34+ cells in the DMD subjects analyzed in this study.
The subjects analyzed in this study were adults (≥ 18 years old) with previously diagnosed diabetes (n = 3522).
Sociodemographic data of the subjected analyzed in this study were presented in one of our previous papers [ 12].
All 297 SA strains analyzed in this study were subjected to phenotypic screening to identify MRSA strains, and 11.78% of all SA carriers (sampled once or multiple times) carried MRSA strains in their nares.
We were unable to match or analyze the subject by gender in this pilot study.
The remaining 504 subjects were analyzed for this study.
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