The phrase "analyzed activation of" is correct and usable in written English.
It can be used in contexts related to research, data analysis, or discussions about processes that involve examining or interpreting activation levels or patterns.
Example: "The researchers analyzed activation of specific brain regions during the cognitive task."
Alternatives: "examined activation of" or "assessed activation of".
Since TLR2 is well known to activate MAPK pathways [32], we analyzed activation of ERK1/2, JNK and p38.
The first paper [ 5] analyzed activation of MAPKs in phorbol ester-activated Jurkat T cells or negatively selected peripheral blood T cells following fixation with 1% paraformaldehyde and permeabilization with 0.1% saponin.
Next we analyzed activation of MAP kinase pathways by BMPs.
To further study tellurite-mediated toxicity we analyzed activation of stress promoters ibpA, sulA, P3rpoH and cspA in response to K2TeO3.
To study the effect of MMP19 in T cell-mediated immune responses we used fluorescein isothiocyanate (FITC) as a hapten in the model of CHS and analyzed activation of DCs and CD8+ T cells.
In the spinal cords of G93A SOD1 mice, caspase-12 is activated in symptomatic period and can be inhibited by overexpression of XIAP (X-linked inhibitor of apoptosis protein [45], [46]. Then, we analyzed activation of caspase-4 (the human orthologue of rodent caspase-12) following tunicamycin treatment.
We have also performed Western blotting to analyze activation of MAPK signaling using anti p-ERK antibody and densitometry analysis.
This methodology has been used to develop two different models of lung adenocarcinoma to analyze activation of pathways downstream of specific mutations and to assess the potential of therapeutic targeting strategies [ 112, 113].
Moreover, we analyzed the activation of some activator protein (AP -1 trAP -1iptranscriptionomponents.
To analyze whether ER-induced apoptosis contributes to excitotoxicity in oligodendrocytes, we analyzed the activation of caspase-12, which resides on the outside of ER membrane and is cleaved and activated during ER stress.
To analyze whether IGF-1 signaling pathway is induced in cells overexpressing IGF-1Ea compared to control cells, cell extracts were analyzed for activation of the downstream mediators of IGF-1 signaling, PDK1 and AKT1.
