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Although this physical association of periostin with the Notch1 precursor might be appropriate, by the Western blot analysis, we detected the N1™ of heterodimeric Notch1 in the wild-type molar PDL, whereas we could not detect its precursor.
In the univariate analysis, we detected the existence of a statistical association only between recent infection and age, with the greatest portions of recently infected among the participants <25 years-old and those 60 years or older (p<0.001, Tables 1 and 2).
To confirm the finding of immunohistochemical (IHC) analysis, we detected the TET2 protein expression in the CRC cell lines.
In this analysis, we detected the binding site of PPAR α by Promo, but not by TRANSFAC.
By the logistic regression analysis, we detected the Ki67 LI, Bax score, and TS score as independent factors (Table 4).
Then, in addition to in silico analysis, we detected the response to LPS or bacteria both in vitro and in vivo.
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In this analysis, we detect the difficulty of learners regarding the violation of constraints.
From this analysis, we detected that only the KEGG2path-Closeness pair satisfied these two criteria (see Additional File 1 and Figure 2).
Additionally, using the resampling simulations analysis, we detected that the upper 11th percentile subset of SNMs in RNA genes (25 mutations), lost its statistical significance making them the RNA genes' SNMs with functionality values most similar to those of disease-causing mutations (supplementary table S6 B, Supplementary Material online).
In this linkage analysis, we detected changes in the detoxification network.
Using reverse transcription-polymerase chain reaction (RT-PCR) and Southern blot analysis, we detected transcripts for the receptor in MCF-7, SK-BR-3 and MDA-MB231 cells.
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