All six genes selected for protein analysis were induced by Th1 cytokines, and for most proteins the expression by atopic dermatitis keratinocytes was significantly lower than for normal skin or psoriasis keratinocytes.
Solid SL2 tumours for cell proliferation analysis were induced by subcutaneous injection of 107 SL2 cells on the chest of mice and analysed after 4 and 10 days.
Many of the genes analysed by Northern analysis were induced at a lower level in the tPA producing culture than in the DTT treated culture or in the strain overexpressing ire1 (Fig. 2).
Furthermore, Wnt-3a and its receptor FZD4, also identified by microarray analysis, were induced both in tamoxifen resistant cells and in Sox2-overexpressing cells, while expression of WNT4, which inhibits Wnt/β-catenin signalling (Elizalde et al, 2011), was reduced (Fig 7C).
Interestingly, unlike many of the 20E-inducible genes we identified by microarray analysis, EcR was induced to a similar extent in PoA-treated Kcellsls, 20E-treated Kc cells and 20E-treated salivary glands.
In Western blot analysis, iNOS was induced in the SN after injection of LPS in a time- and dose-dependent manner.
In our expression analysis, TaCIPK24 was induced in roots and leaves under salt stress (Fig. 4).
Thus, at the particle concentration applied to microscopy analysis, no cytotoxicity was induced.
RT-PCR analysis showed that OsAP77 was induced not only by infection with these pathogens, but also after treatment with SA, INA, H2O2 or ABA.
Expression analysis revealed that ONAC045 was induced by drought, high salt, and low temperature stresses, and abscisic acid (ABA) treatment in leaves and roots.
