The phrase "analysis of this variant" is correct and usable in written English.
It can be used when discussing the examination or evaluation of a specific variant in a study, research, or any analytical context.
Example: "The analysis of this variant reveals significant differences in the genetic makeup compared to the control group."
Alternatives: "examination of this variant" or "evaluation of this variant."
Functional analysis of this variant suggests that it may partially compromise BRCA1 function.
Functional analysis of this variant shows that it confers a loss-of-function mutator phenotype.
Table 3 shows in more detail the analysis of this variant gene in all the models tested (allelic, additive, recessive and dominant).
To gain further structural insight into how the H87V substitution so dramatically alters the dynamical properties of the protein, we carried out a crystallographic analysis of this variant protein at 1.70 Å resolution (Table 1, crystallographic summary).
(E ) Similar SEC-MALS analysis of SAS-52 265 SAS-52 265(F ) plot of andrage molecular weight From SEC-MALS analysis of this variant as a function of on-column plotein cofcentraverage
Notably, four are in the 3′UTRs of PIAS2, WWC3, MT1X, and TBP, and one is exonic (a CAG repeat in FAM157A; see Suppl. Fig. 3 for detailed analysis of this variant).
Although the majority of the studies analyzing this variant found no association with cancer [ 12, 38, 42- 44], some studies reported an association with breast cancer [ 8], basal cell carcinoma in men [ 45] and a recent meta-analysis of this variant in bladder cancer revealed a significant association after controlling for potential bias [ 46].
Our functional analysis of this KCC2 variant in pyramidal neurons of the mammalian cortex, taken together with the co-segregation and genic intolerance analysis discussed below, is strongly suggestive of a role for KCC2-R952H asusceptibilitylity variant in FS, thereby extending the list of other seizure susceptibility genes including GABRD, CACNA1H, HCN2 and SCN9A 17– 19.
In silico analysis of this sequence variant by TESS (www.cbil.upenn.edu/cgi-bin/tess/tess) and by the Genomatix SNP Inspector (www.genomatix.de) revealed a gain of transcription factor binding sites for the human-specific allele ("T") in comparison to the non-human variant ("C"), for factors such as NEUROD1, Upstream stimulating factor ½, E-box factor, TAL-1, DEP2, c-Myc and NF-kappaE2.
