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Principal component analysis demonstrated significant intercenter reproducibility among four centers.
Sensitivity analysis demonstrated a robust model.
Subsequent immunohistochemical analysis demonstrated that i.t.
This analysis demonstrated complete neutralization of TNFα (Supplementary Figure 6D, E).
ChIP analysis demonstrated that both Rad51 and Rad52 are recruited to Flex1 (Fig. 5c).
Gene expression analysis demonstrated that EFEMP1, an extracellular matrix protein, is associated with TMZ-resistant phenotype.
In vitro analysis demonstrated that JAK2V617F is a constitutively active tyrosine kinase.
This analysis demonstrated that TM0415 utilizes PPi but neither ATP nor ADP to generate myo-inositol monophosphate (Fig. 2a).
This analysis demonstrated a highly significant accumulation of CARc bodies in direct adjacency to sinusoidal vessel walls (Fig. 6c).
Northern blot analysis demonstrated expression of the variant mRNAs.
TEM analysis demonstrated the coexistence of Gd2Zr2O7 and LaPO4 phases.
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