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In this context, initial analyses revealed a normal segregation of these lineages in Coro-1A−/− animals but a reduced density of peripheral T lymphocytes, in general agreement with published data on coronin-1A knockout mice [21], [23].
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Immunohistochemical analyses revealed a cytosolic ATIP localization in luminal epithelial cells of normal breast tissue.
In addition, transcriptomic analyses revealed a strong tendency for increased CBX2 mRNA levels in many cancers compared with normal tissues, independently of CDKN2A/B silencing.
Logistic regression analyses revealed a significant moderating effect of exercise.
Immunohistochemical analyses revealed a substantial remodeling of atherosclerotic lesions.
High-throughput analyses revealed a bi-phasic response to lice.
None of these analyses revealed a significant interaction.
Statistical analyses revealed a significant load effect (P < 0.001).
The analyses revealed a mammal specific duplication event for PGK.
Both analyses revealed a marginal structure in three clusters.
Sequence analyses revealed a single nucleotide change inside OAS-A1/OLD3.
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