Exact(10)
That study analysed treatment outcome in 83 patients and showed each subtype to be distinct in their responsiveness to therapy.
The resutling estimates of overall remission and fatality rates were used for the various analysed treatment scenarios.
Despite this primary difference, the results were not stable across the analysed treatment groups particularly in the group with highest pCR rates (taxane and anthracycline treatment).
To include more articles, eligibility criteria would have had to be broader, but increasing the variability of the analysed treatment programs, for instance by including prevention programs, would have lead to even less comparability of results.
Studies of early rheumatoid arthritis (RA) cohorts have analysed treatment response and prognostic factors at fixed time points.
Many studies have analysed treatment retention in OSTM [ 4, 13- 20] or factors associated with retention [ 21, 22].
Similar(50)
Next, expression levels of the determined RA-related genes were analysed following treatment with antiheumatic drugs.
The scores on these items were dichotomised (see above) and analysed by treatment, using the χ-test.
BrdU incorporation was analysed following treatment of HET-1A cells with 0.1, 0.5 and 5 μmol/l MBP-peptide for 24 or 48 h.
Markers of endoplasmic reticulum stress (ERS) were analysed following treatment, and their causal role in mediating persin-induced apoptosis was determined using chemical inhibitors and RNA interference.
We further analysed hormone treatment that started ≤ 50 years of age or > 50 years of age (approximate age of menopause) without statistically significant decreased or increased risks.
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