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As we could not obtain Dmknδ specific antibody, we further analysed the localization of the endogenous Rab5 and the Dmknδ5 by confocal microscopy performed on HeLa cells expressing a GFP-tagged Dmknδ5.
Next, we analysed the localization of the modified sulphamidase enzymes in transfected cells.
Initially, we analysed the localization of Ace in stressed and nonstressed rat brain.
To test this idea, we analysed the localization of YFP MglA+ and YFP MglAG21V in frzlof and frzgof mutants.
In a first set of experiments, we analysed the localization of TRPC3 and NCX1 in TRPC3-TG myocytes by immunocytochemistry.
As both developmental and degenerative processes appear to affect the mutant gars synapse, we next analysed the localization of GlyRSP234KY.
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We next examined the effect of Y-27632 on cell polarization by analysing the localization of F-actin, RhoA, phospho-ERMs, and ICAM-3.
To analyse the localization of filamin in dividing GAPA− cells we perfomed microscopic study using GAPA− GFP-FLN cells treated with nocodazole.
In the current study we have determined the contribution of soluble FtsY to co-translational targeting in vitro and have re-analysed the localization of FtsY in vivo by fluorescence microscopy.
By analysing the localization of three MglA proteins, we established a direct link between MglA/GTP localization and function.
To quantitatively analyse the localization of the reconstruction, we employ the Dice coefficient to judge how well the reconstruction locate the inclusion for the experimental measurements [ 48, 49].
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