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To determine the rationale for these differences in expression patterns, we analysed the expression level of all the co-amplified genes in five xenografted and one fresh human gliomas containing dmins, the sequence of which has been previously analysed in detail [14], [15].
Therefore, we analysed the expression level of apple MLO genes identified in this study in response to the interaction with P. leucotricha.
We analysed the expression level of EIF5A2 in tumours with different thickness and ulceration, as thickness and ulceration are well-known prognostic markers for patients with PM.
As for ABA metabolism, we analysed the expression level of the biosynthetic genes ABA1, ABA2, NCED6 and NCED9, and of the catabolic gene CYP707A2, on dag2 and wild-type seeds imbibed 12 hours in the dark or under R light.
We analysed the expression level of miR-3099 in P19 cells and found a statistically significant (P = 0.04) ~2-fold upregulation of miR-3099 in ~50% differentiated P19 cells compared to the proliferating cells.
Since the expression levels of p53 are difficult to interpret, as in many tumours p53 accumulates as an inactive mutated protein, we also analysed the expression level of Bax.
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To identify miRNAs differentially expressed in NPC, we analysed the expression levels of 270 human miRNAs in 13 NPC and 9 normal nasopharyngeal tissues.
Here, we quantitatively analysed the expression levels of APP, IL-1β, and C1QA and determined the localisation of APP in gingival tissues.
To determine the contribution of both proteins to the repression of MBF-dependent transcription we analysed the expression levels of 14 MBF-dependent transcripts in wt, nrm1Δ and yox1Δ cells (Figure 2B).
To confirm the involvement of proteolytic pathways in drug-induced NANOG and OCT4 degradation, we analysed the expression levels of several key regulatory factors of these pathways in a time course experiment by RT-PCR.
We analysed the expression levels of the four MMR genes among 61 primary MB tumours.
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