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We observed that among altered genes are those encoding the 14-3-3 14-3-3 14-3-3amily of highly conserved proteins.
Correlation in gene expression even among altered genes between the two with the LL allele was no stronger than between the LL allele and SL allele (0.82 vs 0.88; correlation between these and unaffecteds averages 0.65 for comparison).
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Among these, altered plasma fibrinolysis promotes fibrin deposition, which may create microvascular alterations.
But among the altered genes were 18 known to be associated with obesity and another 21 linked with adult-onset diabetes.
Among the altered phenotypes observed, increased leaf angle was one of the most dramatic defective phenotypes.
We then wanted to identify the interacting network among the altered genes in the context of other large biological pathways.
To identify the likely identical regulatory controls among the altered genes, genes common to a single cluster and Gene Ontology term were taken for promoter analysis.
Among the altered genes, ARHGAP26 and MLLT1 have been associated with leukemia-specific translocations, DDHD2, FGFR1 and PTPN1 have tumor-promoting potential in breast cancer, and CTNNA3 may promote tumor formation in urothelial cancer.
Among the altered HOX genes, HOXA5 seems to be the most important.
Among the altered genes, we have more extensively analysed the expression pattern of Hif1α.
Among transcripts altered in expression by space conditions, some were common genes response to abiotic and biotic stress.
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