The phrase "ambient pollutants" is correct and usable in written English.
It can be used when discussing environmental issues, particularly in relation to air quality and the presence of harmful substances in the atmosphere.
Example: "The study focused on the effects of ambient pollutants on respiratory health in urban areas."
Alternatives: "environmental contaminants" or "atmospheric pollutants".
Additional limitations include the absence of indoor home monitoring and the use of fixed-site ambient pollution monitoring, which may result in exposure misclassification, particularly for ambient pollutants that have strong local sources (Briggs et al. 1997).
The association between ambient pollutants and poorer gain of pulmonary volumes in children living in more polluted areas suggests that air pollution in the residence area may be a part of the causal chain of reactions leading to retardation in pulmonary function growth during the preadolescent years.
Through chemical reactions with other ambient pollutants, NOx can form pollutants that are harmful to health, including nitrogen dioxide, ozone, and particles such as ammonium nitrate that contribute to particulate matter (PM) air pollution.
The total impact of NOx emissions on mortality is the impact of both the primary pollutants emitted directly by vehicles and the secondary pollutants formed through chemical reactions between primary pollutants and other ambient pollutants.
A number of ambient pollutants have been involved, including carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3) and particulate matter (PM).
KCL also notes that diet is believed to help protect against pollution: "A study conducted in Mexico City has shown that children eating more antioxidants, from fresh fruit and vegetables, are better protected against the oxidative effects of ozone and other ambient pollutants".
Most pulmonary immunotoxicology studies of ambient pollutants have been broadly designed to discern if overall humoral or cell-mediated immunity (CMI) was altered; few have assessed effects on particular aspects of immune function.
The API compares five main air pollutants, i.e. sulfur dioxide (SO2), respirable suspended particulates (RSP), nitrogen dioxide (NO2), carbon monoxide (CO) and Ozone (O3) as sub-indexes, which are calculated separately from a segmented linear function that transforms ambient pollutants concentrations into a normalized scale extending from 0 to 500.
Second, as atmospheric composition changes over a finite length of time, ambient pollutants levels and meteorological variables (i.e., temperature and wind) can be viewed as a data signal with a low rate of change.
In the last decades, ambient pollutants, combustion generated particles and polycyclic aromatic hydrocarbons associated with them have been identified as health problems with substantial impact on our immune responses [3] [5].
While in agreement with cross-sectional findings and animal studies, these results need to be corroborated with longitudinal studies particularly designed to evaluate this hypothesis, and to investigate the role of endogenous and exogenous co-factors that may interact with the vascular toxicity of ambient pollutants.
