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Air pollution epidemiologic studies use ambient pollutant concentrations as surrogates of personal exposure.
Having demonstrated an effect of ambient pollutant exposure on baseline systemic levels of IL-6, future research should focus on whether ambient pollution exposure modifies the effect of inflammatory responses to controlled pollution exposures human chamber studies.
Conclusion: Measured and estimated exposures from ambient pollutant monitors and LUR model suggest associations between autism and prenatal air pollution exposure, mostly related to traffic sources.
Conclusions: This study demonstrates a significant association between ambient pollutant levels and baseline levels of systemic IL-6.
Data were obtained from microenvironmental concentration measurements, integrated, 24-h personal measurements, indoor and outdoor pollutant monitors at the participants' residences, from ambient pollutant monitors at fixed-site locations in each city, and from real-time diaries and questionnaires completed by the technicians and participants.
Exposure to ambient pollutant particle (APP) is associated with increased cardiovascular morbidity and mortality.
Thus, participants' exposures to ambient pollutant might be misclassified.
Using ecological data to assume ambient pollutant concentrations as surrogates of individual exposure adds error.
Similar(3)
The total impact of NOx emissions on mortality is the impact of both the primary pollutants emitted directly by vehicles and the secondary pollutants formed through chemical reactions between primary pollutants and other ambient pollutants.
Through chemical reactions with other ambient pollutants, NOx can form pollutants that are harmful to health, including nitrogen dioxide, ozone, and particles such as ammonium nitrate that contribute to particulate matter (PM) air pollution.
A number of ambient pollutants have been involved, including carbon monoxide (CO), nitrogen dioxide (NO2), sulfur dioxide (SO2), ozone (O3) and particulate matter (PM).
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