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The altitude threshold was introduced because the ISIS-1 data at lower altitudes show discrepancies with several of the other data sets.
There is no specific altitude threshold that can be considered safe for everyone and below which no one will develop altitude DCS.
The fact that recombinant Epo boosts maximal oxygen transport at sea level but not at high altitude implies the existence of an "altitude threshold" beyond which the ergogenic effect of rhEpo on V̇O2max evanishes.
In order to gain insights into the altitude threshold beyond which the effect of rhEpo on maximal oxygen uptake becomes marginal, seven of the eight subjects (subjects #1 to #7) underwent a non-invasive experiment on two separate occasions, i.e. before and after five weeks of rhEpo treatment.
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In 25%% of the profiles, two distinct scale heights were necessary, with a scale height on the order of several tens of kilometers changing above a threshold altitude to a scale height on the order of 100 km.
As the altitude increases, the threshold velocity for sand movement linearly increases with altitude, and the sand transport per unit width decreases gradually.
This implies that there should be a threshold altitude at which V̇O2max is less dependent on CaO2.
This finding implies that there is a threshold altitude between 3500 and 4500 m, from which little or no benefit should be expected from an increased hemoglobin concentration.
20 In the 1980s, the same research laboratory revised the threshold altitude for aerobic impairment down to 1219 m.
If a threshold altitude is selected to attempt to separate the snow-covered tops from the lower portions of the mountain, compromises are required.
Based on these data, Terrados et al 22 theorised that the threshold altitude for aerobic impairment may not be universal for all, and their group ultimately showed that trained athletes demonstrate a reduction in VO2max at a much lower threshold altitude (900 m) than untrained individuals (1200 m).
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