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By parametrically altering the duration of each trial we obtained strong evidence that exposure time (PAT) itself directly modulates amygdala activation.
As fast transmission to excitatory motor neurons is mediated by nicotinic acetylcholine receptors [34], [35], [36] this predicts that sub-maximal concentrations of nicotinic antagonists will decrease the duration of active episodes without altering the duration of quiescent episodes.
Activity modification at an individual level such as altering the duration, intensity, and location where individuals are physically active can also help reduce air pollution exposure and dose.
Altering the duration of exercise was critically important to maximization of negative geotaxis ability across ages, whereas altering the number of exercise repetitions did not change the outcome significantly (Table 1, hours of exercise-by-age, p<.0001).
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The vertical location of the drops determined their relative pitch, and changing their size altered the duration of each note.
Changing the plasmid design, from a cytomegalovirus (CMV) to an ubiquitin C (UbC) promoter substantially altered the duration of expression.
In the intersensory conditions, additional contextual cues may alter the duration of perception when combined across sensory modalities.
The strongest evidence thus far obtained comes from the manipulation of male-specific neurons expressing the GR68A gustatory receptor protein, found in gustatory sensilla on fore-tarsi, which apparently altered the duration of male courtship of females [10].
Adjusting the contributions of fast and slow release will alter the duration of postsynaptic responses.
Different treatments were created to alter the duration of male – female interaction.
Hence, functional changes in microcephaly genes could conceivably alter the duration of symmetric divisions to ultimately change the number of neurons produced during brain development.
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CEO of Professional Science Editing for Scientists @ prosciediting.com