The phrase "altered vessel" is correct and usable in written English.
It can be used in contexts related to changes made to a container or a ship, often implying modifications for a specific purpose.
Example: "The altered vessel was equipped with advanced navigation systems to enhance its performance in rough seas."
Alternatives: "modified container" or "changed ship".
However, modified expression of ADAMTS1 did not influence the degree of pericyte coverage, indicating that altered vessel stabilization by pericytes is not involved in the altered blood vessel morphology observed.
Barbels allowed to regenerate for longer than 7 days also had altered vessel morphologies, but did not depart from this basic regenerative pattern.
At an early time-point (day 14), loss of CD34 on non-hematopoietic cells affected tumor vasculature resulting in decreased tumor size, increased vascular leakage and altered vessel morphology.
In support of the second hypothesis, we found increased tissue carbocyanine leakage, altered vessel morphology and increased tumor cell extravasation in the lungs of Cd34−/− mice, compared to wildtype animals.
Up-regulation of inflammatory mediators and adhesion molecules are early features of diabetic retinopathy[6], leading to accumulation of leukocytes, altered vessel reactivity and subsequent activation of receptors and transcription factors, ultimately resulting in apoptosis or proliferation of various cell types in the retina[5], [7], e.g., loss of pericytes and proliferation of endothelial cells.
In addition, signs of vascular degeneration, abnormal vascular smooth muscle function, and altered vessel compliance have been described in humans chronically and acutely exposed to toxic lead levels.
In one of our previously reported studies on the same cohort a third of the investigated CC showed altered vessels with a discontinuous, hypoplastic muscle wall layer, which could reflect immature tumor vascular entities possibly in the course of arteriogenesis [ 16].
Results also showed how variations in the mechanisms of cellular persistence alter vessel growth.
Furthermore, abnormal vessel wall function with loss of tensile forces and increased intramural pressure may alter vessel integrity increasing tortuosity.
Hypoxia and macrophages within atherosclerotic plaques may promote intraplaque and intra vessel wall angiogenesis and alter vessel wall microcirculation [ 36].
These alter vessel diameter by promoting either smooth muscle relaxation, which increases vessel diameter, or smooth muscle contraction, which decreases vessel diameter (reviewed by Attwell et al., 2010).
