The phrase "altered cells to" is correct and usable in written English.
It can be used in contexts related to biology, genetics, or any field discussing modifications to cells.
Example: "The researchers altered cells to enhance their resistance to disease."
Alternatives: "modified cells to" or "changed cells to".
However, current in vivo imaging techniques lack the ability to track the real time migration and homing of genetically altered cells to cancer tissue with acceptable resolution.
The known decades-long latency of MPM then suggests that there is ample time for appreciable fields of clonally altered cells to accumulate, perhaps leading to malignancy through a combination of acquired genetic and epigenetic alterations enhanced by repeated mitotic selection.
Consequently, it is important to note that the induction of TRIM29 may allow some altered cells to survive.
An increased rate of production for a short time during early life will provide a larger number of altered cells to be acted upon by other factors in the future, and will therefore appreciably affect the incidence at, say, age 60.
The same increased rate of production of the first change applied for the same short period during middle age will, on the other hand, have little effect on the incidence at age 60, since there will be but little time left for the altered cells to be acted upon.
Moreover, possible structural changes in the vasculature of SHR and altered cell-to-cell communication may both participate in altered vascular function observed in SHR rats.
This included altered cell-to-cell signaling and cell-to-cell interaction, perturbations in lipid metabolism, glycosylation, an array of post translational modifications as well as undue regulation of development and cellular movement programs [8].
Binding of Ephrin to the EphR may cause activation and deactivation of the Eph and Ephrin downstream signaling cascades resulting in altered cell-to-cell interactions and cell migration.
One of the key molecular examples of NOA in human breast carcinomas is the association between the EMT phenotype and altered cell-to-cell adhesion due to reduced CDH1 expression [ 39, 42].
Hence, downregulation of Pcdh10 gene expression observed in this study is an index of altered cell-to-cell communication, which may also signify disturbances in synaptic or neuronal plasticity within the striatal complex.
Some scientists are also pursuing treatments based on a related technique known as optogenetics, which involves genetically altering cells to restore light sensitivity to the retina.
