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On the basis of the paracrystalline layered model of the perfluorinated membrane nanostructure these structure alterations were interpreted as an increase in the thickness of the liquid layers (ionic channels) separating clusters of ionomer groups in MF-4SK membranes.
The histopathological CNS alterations were interpreted as follows: +++: severe; ++: moderate; +: slight; -: none.
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The observed alterations are interpreted in the light of ion solid interaction, involving the thermodynamic stability and the valence state of the oxides.
Both effects were interpreted by alterations in cell cycle regulation and not by action of apoptosis [ 17].
The MR imaging method as described in their previous publication [28] allowed them to identify alterations of the junctional zone that were interpreted as signs of focal and diffuse adenomyosis, respectively, according to the guidelines provided by Reinhold et al. [29].
Alterations coinciding with known polymorphic CNV regions were interpreted as benign and not included in the analysis report.
In early studies of the role of catecholaminergic neurons in ICSS [23], [50], drug-induced changes in behavior were interpreted in terms of such alterations in sensitivity.
No trends were interpreted.
The detection of a variant multiple times in patients with Morquio A from independent families strongly supports linking a variant with Morquio A disease; conversely, novel, difficult-to-interpret gene alterations (e.g., many missense alterations) should be interpreted with caution and only limited conclusions drawn [Morrone et al., 2014].
These alterations may be interpreted as a switch from the transmethylation pathway to the transsulfuration pathway.
These alterations could be interpreted as premature megakaryocytic differentiation and/or activation of K562WASPKO cells.
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CEO of Professional Science Editing for Scientists @ prosciediting.com