The phrase "alterations of mitochondrial" is correct and usable in written English.
It can be used in contexts discussing changes or modifications related to mitochondria, often in scientific or medical discussions.
Example: "The study focused on the alterations of mitochondrial function in aging cells."
Alternatives: "changes in mitochondrial" or "modifications of mitochondrial".
The mutant DN22-CB1 receptor is functional, but it does not mediate cannabinoid-induced alterations of mitochondrial activity and mobility.
Mitochondrial permeability transition pore (mPTP) plays a central role in alterations of mitochondrial structure and function leading to neuronal injury relevant to aging and neurodegenerative diseases including Alzheimer's disease (AD).
Therefore, alterations of mitochondrial function caused by severe ischemia may reduce the accumulation of MIBI within the cells [26].
Mitophagy can be induced by nutrient deprivation, oxidative stress, hypoxia, mitochondrial dysfunction, and alterations of mitochondrial permeability transition (MPT) (Lemasters, 2005).
One potential explanation for this result is that PA28γ is a non-ATP dependent regulator of the UPS [36], and therefore less connected to alterations of mitochondrial metabolism.
Interestingly, alterations of mitochondrial activity occurring during aging were associated with an important reduction in MyHC-I, IIx and IIb expression.
Another type of intervention is a specific alteration of mitochondrial electron transport that alters the generation of reactive oxygen species (ROS) [ 15, 16].
To assess whether a possible alteration of mitochondrial biogenesis or degradation results in altered levels of mitochondria, we quantified marker proteins of the mitochondrial inner and outer membrane from aged Pink1−/− brain with immunoblots.
To determine whether H2O2-induced oxidative stress alters mitochondrial bioenergetics in ONH astrocytes and whether CoQ10 treatment inhibits the alteration of mitochondrial bioenergetics, we measured cell viability, ROS generation and cellular ATP level.
These genes code for two enzymes involved in fatty-acid metabolism, and we have demonstrated in human cells that the HSP pathophysiology includes alteration of mitochondrial architecture and bioenergetics with increased oxidative stress.
Alteration of mitochondrial metabolism due to ID may be responsible for these observations.
